Modified Citrus Pectin

Fighting Cancer Metastasis and Heavy Metal Toxicities with Modified Citrus Pectin

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This Supplement Can Benefit Almost Every Cancer

Modified Citrus Pectin Highlights:

Pectin is a complex carbohydrate that is abundantly present in citrus fruits. Modified citrus pectin (MCP) is composed of short, non-branched carbohydrate chains derived from the peel and pulp of citrus fruits.
Compelling research suggests that modified citrus pectin may help block the growth and metastasis of solid tumors such as breast, colon, and prostate cancers.
Intriguing clinical studies suggest that supplementation with MCP stabilizes disease progression and lengthens PSA doubling times in men with prostate cancer.
Modified citrus pectin may represent a safe, non-toxic detox of chelating toxic metals—without the need for intravenous infusions.
Supplementation with MCP has been shown to increase excretion of dangerous metals such as mercury, arsenic, lead, and cadmium—without removing essential minerals like calcium, magnesium, and zinc from the body. Start by using BioSil. Arsenic is what is known as a trivalent heavy metal. It is very difficult to remove from the body unless silica, found in BioSil, is used to mobilize it from tissues. Once it is mobilized, the modified citrus pectin in PectaClear bind to the arsenic (and other metals and chemicals) and remove it from the body with no detox symptoms. Using this method, it will take two years to completely remove arsenic from the body.
A clinical study showed that supplementation with an MCP-alginate complex reduced total body toxic heavy metal burden in patients with a variety of health concerns.
MCP is considered safe and well tolerated. Dosages range from 6 to 30 grams per day in divided dosages; a typical dose is 5 grams three times daily.

Per Sloan-Kettering:

Uses

Diarrhea

Pectin has been shown to be effective in treating diarrhea in children with persistent diarrhea.

Hypercholesterolemia

Diets supplemented with fiber can benefit patients with high blood cholesterol.

Cancer prevention and treatment

Laboratory and animal studies have shown that pectin has anticancer properties. Human studies are needed to verify these results.

Clinical Summary

Pectin is a soluble fiber rich in galactoside residues. It is present in most plantsbut is concentrated in the peel and pulp of citrus fruits and in apples. Pectin is used as a gelling agent in food. It is also used as a remedy for diarrhea, to lower cholesterol, or to improve satiety. Modified Citrus Pectin (MCP) is an altered form of pectin that has shorter carbohydrate chains and is claimed to be better absorbed by the body.

Preclinical studies suggest inhibitory effects with pectin in colon , breast , liver , and lung cancer cell lines. Preliminary clinical data suggest that pectin is beneficial in treating diarrhea and in reducing levels of toxic heavy metals . Pectin is also effective against hypercholesterolemia , but a recent study showed that source and type of pectin influences its activity . Another study also demonstrated that different types pectin are varied in their properties and may affect appetite and energy intake differently.

Pectin has been promoted for reducing colon cancer risk and damage from radiation therapy, but there is insufficient scientific evidence to support these uses. In a small non-randomized study, MCP was shown to increase the doubling time of prostate-specific antigen in patients with prostate cancer .

Gastrointestinal side effects such as abdominal cramps and diarrhea have been reported with use of pectin.

Mechanism of Action

Modified Citrus Pectin (MCP) acts as a ligand for galectin-3, which plays a major role in tumor formation and progression . Binding of MCP to galectin-3 was shown to inhibit tumor growth, angiogenesis, and metastasis in vivo. MCP is thought to render galectin-3 incapable of binding its receptors that would result in angiogenesis . Galectin-3 is also found in prostate cancer cells and prostate tissue. In another study, MCP was shown to increase doubling time of prostate-specific antigen (PSA), presumably by binding galectin-3 . Pectin induced apoptosis in adenocarcinoma cells in vitro via caspase-3 activity resulting in DNA degradation.